Please use this identifier to cite or link to this item: https://biore.bio.bg.ac.rs/handle/123456789/72
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dc.contributor.authorĐorđević, Jelenaen_US
dc.contributor.authorJasnić, Nebojšaen_US
dc.contributor.authorVujović, Predragen_US
dc.contributor.authorLakić, Ivaen_US
dc.contributor.authorĐurašević, Sinišaen_US
dc.contributor.authorGavrilović, Ljubicaen_US
dc.contributor.authorCvijić, Gordanaen_US
dc.date.accessioned2019-06-18T12:25:47Z-
dc.date.available2019-06-18T12:25:47Z-
dc.date.issued2012-02-01-
dc.identifier.issn0931-2439-
dc.identifier.urihttps://biore.bio.bg.ac.rs/handle/123456789/72-
dc.description.abstractThe heart is an organ especially sensitive to the sympathetic overstimulation and therefore to the influence of stressors and hypertension. The aim of the present study was to investigate the effect of two distinct types of stressors, acute immobilization (2h) and chronic isolation stress (21days), as well as their combined effect on the activity of monoamine oxidase (MAO), superoxide dismutase, catalase (CAT) and the ascorbic acid (AA) content in the heart of normotensive and spontaneously hypertensive rats (SHR). The results obtained show that in basal conditions heart MAO and CAT activity (p<0.05), as well as AA concentration (p<0.01) were higher in SHR than in normotensive ones. The acute immobilization significantly decreased heart MAO activity in both examined strains (p<0.01). On the other hand, chronic isolation, separately or in combination with immobilization, did not affect this enzyme, in the heart of either hypertensive or normotensive rats, which was associated with the reduced antioxidative protection (p<0.01, p<0.05).en_US
dc.language.isoenen_US
dc.relation.ispartofJournal of Animal Physiology and Animal Nutritionen_US
dc.subjectAntioxidative protectionen_US
dc.subjectHearten_US
dc.subjectMAOen_US
dc.subjectSHRen_US
dc.subjectStressen_US
dc.titleDistinct and combined effects of acute immobilization and chronic isolation stress on MAO activity and antioxidative protection in the heart of normotensive and spontaneously hypertensive ratsen_US
dc.typeArticleen_US
dc.identifier.doi10.1111/j.1439-0396.2010.01122.x-
dc.identifier.pmid21244524-
dc.identifier.scopus2-s2.0-84855731729-
dc.identifier.urlhttps://api.elsevier.com/content/abstract/scopus_id/84855731729-
item.languageiso639-1en-
item.cerifentitytypePublications-
item.openairetypeArticle-
item.fulltextWith Fulltext-
item.grantfulltextrestricted-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
crisitem.author.deptChair of Comparative Physiology and Ecophysiology-
crisitem.author.deptChair of Comparative Physiology and Ecophysiology-
crisitem.author.deptChair of Comparative Physiology and Ecophysiology-
crisitem.author.deptChair of Comparative Physiology and Ecophysiology-
crisitem.author.deptChair of Comparative Physiology and Ecophysiology-
crisitem.author.orcid0000-0002-6510-1027-
crisitem.author.orcid0000-0003-0333-333X-
crisitem.author.orcid0000-0002-9444-4758-
crisitem.author.orcid0000-0001-8894-7300-
crisitem.author.orcid0000-0003-4406-8376-
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