Please use this identifier to cite or link to this item: https://biore.bio.bg.ac.rs/handle/123456789/4019
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dc.contributor.authorVasiljević, Anaen_US
dc.contributor.authorBursać, Biljanaen_US
dc.contributor.authorDjordjevic, Anaen_US
dc.contributor.authorMilutinović, Danijela Vojnovićen_US
dc.contributor.authorNikolić, Marinaen_US
dc.contributor.authorMatić, Gordanaen_US
dc.contributor.authorVeličković, Natašaen_US
dc.date.accessioned2021-04-16T15:41:51Z-
dc.date.available2021-04-16T15:41:51Z-
dc.date.issued2014-
dc.identifier.issn1436-6207-
dc.identifier.issn1436-6215-
dc.identifier.urihttps://biore.bio.bg.ac.rs/handle/123456789/4019-
dc.description.abstractHigh fructose consumption provokes metabolic perturbations that result in chronic low-grade inflammation and insulin resistance. Glucocorticoids, potent anti-inflammatory hormones, have important role in pathogenesis of diet-induced metabolic disturbances. The aim of this study was to examine the link between glucocorticoid metabolism and inflammation in the liver of fructose-fed rats.en_US
dc.language.isoenen_US
dc.relation.ispartofEuropean Journal of Nutritionen_US
dc.relation.ispartofseries53;1393-1402-
dc.titleHepatic inflammation induced by high-fructose diet is associated with altered 11βHSD1 expression in the liver of Wistar ratsen_US
dc.typeArticleen_US
dc.identifier.doi10.1007/s00394-013-0641-4-
dc.identifier.pmid24389792-
item.languageiso639-1en-
item.cerifentitytypePublications-
item.openairetypeArticle-
item.fulltextWith Fulltext-
item.grantfulltextrestricted-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
crisitem.author.deptChair of Biochemistry and Molecular Biology-
crisitem.author.orcid0000-0002-0142-1056-
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