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Please use this identifier to cite or link to this item: https://biore.bio.bg.ac.rs/handle/123456789/1457
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dc.contributor.authorKnežević Vukčević, Jelenaen_US
dc.contributor.authorSimić, D.en_US
dc.date.accessioned2019-09-26T11:38:20Z-
dc.date.available2019-09-26T11:38:20Z-
dc.date.issued1991-01-01-
dc.identifier.issn0300-9084-
dc.identifier.urihttps://biore.bio.bg.ac.rs/handle/123456789/1457-
dc.description.abstractThe repair response of Escherichia coli K-12 to bleomycin was examined in Rec- mutants showing differential sensitivity to this agent. Sedimentation analysis of the cellular DNA showed incision after bleomycin treatment. The subsequent reformation of the DNA, found in the wild-type and the recD mutant, was abolished in the recB and delayed in the recF and recBC sbcB mutants. The bleomycin-induced SOS response was reduced in strains containing recB or recBC sbsB mutations. It is suggested that the RecBCD pathway has the main role in the efficient repair of bleomycin-induced DNA damage. © 1991.en_US
dc.language.isoenen_US
dc.relation.ispartofBiochimieen_US
dc.subjectbleomycinen_US
dc.subjectrepair of DNA strand breaksen_US
dc.subjectrecombination deficiencyen_US
dc.titleRecBC promoted repair of bleomycin damage in Escherichia colien_US
dc.typeArticleen_US
dc.identifier.doi10.1016/0300-9084(91)90118-K-
dc.identifier.pmid1716995-
dc.identifier.scopus2-s2.0-0025905609-
dc.identifier.urlhttps://api.elsevier.com/content/abstract/scopus_id/0025905609-
item.openairetypeArticle-
item.cerifentitytypePublications-
item.grantfulltextnone-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.languageiso639-1en-
item.fulltextNo Fulltext-
crisitem.author.deptChair of Microbiology-
crisitem.author.orcid0000-0002-8138-6579-
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